Real Science: MIT Researchers Collapse Corona Virus Shell with Resonant Frequencies

Ultrasound scans like those used to track the growth of a foetus can destroy coronavirus cells by forcing their surface to split apart and implode, new research suggests.

MIT researchers conducted a mathematical analysis based on the physical properties of generic coronavirus cells.

It revealed medical ultrasound scans may be able to damage the virus’s shell and spikes, leading to collapse and rupture.

Ultrasounds are already used as a treatment for kidney stones but the MIT team are calling for further research on its viability as a treatment for Covid-19. Click here for article.

Pseudoscience: Fake Papers in Medical Journals

We previously noted that over half the papers in medical journals cannot be replicated in pharmaceutical company laboratories. This is the result of a combination of bad science and fake science.

Red Flags Raised Over Chinese Research Published in Global Journals

Apparently fraudulent data in dozens of peer-reviewed articles spark fresh worries about ‘paper mills’ used by researchers under pressure to publish

Wall Street Journal by Eva Xiao July 5, 2020 5:00 am ET

HONG KONG—Internationally peer-reviewed journals published more than 100 scientific research papers from China-based authors that appear to have reused identical sets of images, raising questions about the proliferation of problematic science as institutions fast-track research during the coronavirus pandemic.

The cache of 121 papers, credited to researchers from hospitals and medical universities across roughly 50 cities in China, all shared at least one image with another—a sign that many were likely produced by the same company or “paper mill,” said Elisabeth Bik, a California-based microbiologist and image-analysis expert who identified the trove.

Though the reports were published by different authors over a four-year period, many included identical snapshots of cell colonies—sometimes rotated or cropped to appear unique—despite focusing on different research topics. Phrases in figure captions were also repeated verbatim across the collection of reports.

Vaccine Contaminants Plague the Industry

Every Human Vaccine Tested Was Contaminated by Unsafe Levels of Metals and Debris Linked to Cancer and Autoimmune Disease, New Study Reports

Researchers examining 44 samples of 30 different vaccines found dangerous contaminants, including red blood cells in one vaccine and metal toxicants in every single sample tested – except in one animal vaccine.
Using extremely sensitive new technologies not used in vaccine manufacturing, Italian scientists reported they were “baffled” by their discoveries which included single particles and aggregates of organic debris including red cells of human or possibly animal origin and metals including lead, tungsten, gold, and chromium, that have been linked to autoimmune disease and leukemia.

In the study (http://medcraveonline.com/IJVV/IJVV-04-00072.pdf), published this week in the International Journal of Vaccines and Vaccination, the researchers led by Antonietta Gatti (http://ec.europa.eu/health/ph_risk/committees/scmp/documents/cv_gatti_en.pdf), of the National Council of Research of Italy and the Scientific Director of Nanodiagnostics (http://www.nanodiagnostics.it/en/), say their results “show the presence of micro- and nano-sized particulate matter composed of inorganic elements in vaccine samples” not declared in the products’ ingredients lists.

Lead particles were found in the cervical cancer vaccines, Gardasil and Cervarix, for example, and in the seasonal flu vaccine Aggripal manufactured by Novartis as well as in the Meningetec vaccine meant to protect against meningitis C.

Samples of an infant vaccine called Infarix Hexa (against diphtheria, tetanus, pertussis, hepatitis B, poliomyelitis and haemophilus influenzae type B) manufactured by GlaxoSmithKline was found to contain stainless steel, tungsten and a gold-zinc aggregate.

Other metal contaminants included platinum, silver, bismuth, iron, and chromium. Chromium (alone or in alloy with iron and nickel) was identified in 25 of the human vaccines from Italy and France that were tested.

Real Science: Using Ultrasound to Kill Cancer

The leading physics labs are publishing papers on using frequencies to kill cancer. Here are two from Cal Tech and Cold Springs National Laboratory. These studies are published in the leading physics journals, not the medical journals. It will be decades before the average physician takes advantage of this.

A Dynamical Model of Oncotripsy by Mechanical Cell Fatigue: Selective Cancer Cell Ablation by Low-Intensity Pulsed Ultrasound (LIPUS)

E. F. Schibber, D. R. Mittelstein, M. Gharib, M. G. Shapiro, P. P. Lee, M. Ortiz (Submitted on 27 Nov 2019)

The method of oncotripsy, first proposed in [S. Heyden and M. Ortiz (2016). Oncotripsy: Targeting cancer cells selectively via resonant harmonic excitation. Journal of the Mechanics and Physics of Solids, 92:164-175], exploits aberrations in the material properties and morphology of cancerous cells in order to ablate them selectively by means of tuned low-intensity pulsed ultrasound (LIPUS). We propose a dynamical model of oncotripsy that follows as an application of cell dynamics, statistical mechanical theory of network elasticity and ‘birth-death’ kinetics to describe processes of damage and repair of the cytoskeleton. We also develop a reduced dynamical model that approximates the three-dimensional dynamics of the cell and facilitates parametric studies, including sensitivity analysis and process optimization. We show that the dynamical model predicts—and provides a conceptual basis for understanding—the oncotripsy effect and other trends in the data of [D. R. Mittelstein, J. Ye, E. F. Schibber, A. Roychoudhury, L. T. Martinez, M. H. Fekrazad, M. Ortiz, P. P. Lee, M. G. Shapiro, M. Gharib (2019). Selective Ablation of Cancer Cells with Low Intensity Pulsed Ultrasound. BioRxiv] for cells in suspension, including the dependence of cell-death curves on cell and process parameters.

Selective Ablation of Cancer Cells with Low Intensity Pulsed Ultrasound

David R. Mittelstein, Jian Ye, Erika F. Schibber, Ankita Roychoudhury, Leyre Troyas Martinez, M. Houman Fekrazad, Michael Ortiz, Peter P. Lee, Mikhail G. Shapiro, Morteza Gharib
doi: https://doi.org/10.1101/779124Now published in Applied Physics Letters doi: 10.1063/1.5128627

ABSTRACT
Ultrasound can be focused into deep tissues with millimeter precision to perform non-invasive ablative therapy for diseases such as cancer. In most cases, this ablation uses high intensity ultrasound to deposit non-selective thermal or mechanical energy at the ultrasound focus, damaging both healthy bystander tissue and cancer cells. Here we describe an alternative low intensity pulsed ultrasound approach that leverages the distinct mechanical properties of neoplastic cells to achieve inherent cancer selectivity. We show that when applied at a specific frequency and pulse duration, focused ultrasound selectively disrupts a panel of breast, colon, and leukemia cancer cell models in suspension without significantly damaging healthy immune or red blood cells. Mechanistic experiments reveal that the formation of acoustic standing waves and the emergence of cell-seeded cavitation lead to cytoskeletal disruption, expression of apoptotic markers, and cell death. The inherent selectivity of this low intensity pulsed ultrasound approach offers a potentially safer and thus more broadly applicable alternative to non-selective high intensity ultrasound ablation.

Flourescent Microscopy: How to Tell a Frequency is Killing a Virus

The smallest virus is about 20 nanometers and you must see the live virus to determine whether a frequency destroys it. Electron microscopes won’t work as they only operate on dead tissue. A hundred years ago, Royal Rife invented a microscope with about 20,000x resolution on live organisms and determined what frequencies would kill various pathogens.

Today, Leica sells microscopes that can visualize a 20 nanometer organism inside a living cell so anyone with such a microscope can determine what frequencies work best.

Today, there are research groups showing the exact mechanisms on how frequencies can inactivate viruses reporting in leading journals like Nature. See Efficient Structure Resonance Energy Transfer from Microwaves to Confined Acoustic Vibrations in Viruses.

For 20 years this blog has been pointing out that frequency medicine is the future and the future is now.

Mechanisms and therapeutic effectiveness of pulsed electromagnetic field therapy in oncology

Maria Vadalà, Julio Cesar Morales‐Medina, […], and Tommaso IannittiVadalà M, Morales-Medina JC, Vallelunga A, Palmieri B, Laurino C, Iannitti T. Mechanisms and therapeutic effectiveness of pulsed electromagnetic field therapy in oncology. Cancer Med. 2016;5(11):3128–3139. doi:10.1002/cam4.861

Abstract

Cancer is one of the most common causes of death worldwide. Available treatments are associated with numerous side effects and only a low percentage of patients achieve complete remission. Therefore, there is a strong need for new therapeutic strategies. In this regard, pulsed electromagnetic field (PEMF) therapy presents several potential advantages including non‐invasiveness, safety, lack of toxicity for non‐cancerous cells, and the possibility of being combined with other available therapies. Indeed, PEMF stimulation has already been used in the context of various cancer types including skin, breast, prostate, hepatocellular, lung, ovarian, pancreatic, bladder, thyroid, and colon cancer in vitro and in vivo. At present, only limited application of PEMF in cancer has been documented in humans. In this article, we review the experimental and clinical evidence of PEMF therapy discussing future perspectives in its use in oncology. Keywords: Cancer, electromagnetic therapy, oncology, pulsed electromagnetic fields, tumor‐specific frequencies

Anthony Holland’s Magic Frequency

Heart Disease: Causes and Prevention

There a many types of cardiovascular disease (see Medical News Today):

Congenital heart disease

Arrhythmia

Arrhythmia is an irregular heartbeat.

Coronary artery disease

The coronary arteries supply the heart muscle with nutrients and oxygen by circulating blood. Coronary arteries can become diseased or damaged, usually because of plaque deposits that contain cholesterol.

Dilated cardiomyopathy

This is also known as a heart attack, cardiac infarction, and coronary thrombosis. An interrupted blood flow damages or destroys part of the heart muscle.

Heart failure

Also known as congestive heart failure, heart failure occurs when the heart does not pump blood around the body efficiently.

Hypertrophic cardiomyopathy

This is a genetic disorder in which the wall of the left ventricle thickens, making it harder for blood to be pumped out of the heart.

Mitral regurgitation

Also known as mitral valve regurgitation, mitral insufficiency, or mitral incompetence, this occurs when the mitral valve in the heart does not close tightly enough.

Mitral valve prolapse

The valve between the left atrium and left ventricle does not fully close, it bulges upwards, or back into the atrium.

Pulmonary stenosis

It becomes hard for the heart to pump blood from the right ventricle into the pulmonary artery because the pulmonary valve is too tight.

There appear to be two primary causes, other than genetic disorder, for all categories of heart disease.

Diseases related to heart function appear to be caused by infection of the heart, primarily with the same organism that causes cancer. This organism can be clearly seen in an Ergonom 700 microscope that magnifies 25000x. Frequencies for elimination of this organism are documented on this site.
Diseases related to clogging of the arteries are primary the result of nanobacteria. CT scan calcium scores indicate level of arterial obstruction. Nanobacteria form calcium shells which block the arteries. A strategy for reducing calcium scores on a CT scan by 87.5% is documented on this site.

In 2016, 32.3% of deaths were caused by heart disease and 16.3% of deaths were caused by cancer. We estimate that 80% of these deaths could be prevented with frequencies.

Today there are clinical trials using frequency devices, many government approved frequency tools, and thousands of research papers published in PubMed. Eventually health care providers will start bringing these tools into their practice.

Carcinogenesis: A multihit, multistage phenomenon

UPDATE: Cancer is a disease of the mitochondria. Infection of mitochondria by the Rife BX BY virus (DNA sequenced to be bacillus licheniformis) which causes the cell to flip into anerobic energy generation cycle. If a carcinogen is present the cell starts to mutate. After four steps the cell moves into uncontrollable growth. See Tripping Over the Truth by Travis Christofferson.

These bacillus licheniformis organisms can be seen in a 25000x microscope and are photographed in detail in this video. They grow much faster in a solution with carbohydrates, which explains why ketogenic diets help. The researchers using the high power microscopes assert that all saccharides much be removed from the diet of cancer patients, or they are at risk of death from these microbes.

The cancer microbes have many strains and frequencies for all identified strains can be found on this web site. Eliminating these microbes has eliminated cancer cells in some researchers with biopsied tumors. From microscopic photos it can be determine that these microbes cause malfunction of heart cells. This is combination with nanobacteria clogging arteries seems to be the root of heart disease.

If these organisms infect liver cell mitochondria they can cause fatty liver syndrome. If they infect pancreas mitochondria they can cause diabetes.

The multihit research can be found in: Sutherland, Jeff and Bailar, John. The multihit model of carcinogenesis: etiologic implications for colon cancer. J Chronic Dis 1984;37(6):465-80.

While this work built on the research of many of the leading scientists earlier in the 20th century, it marked the beginning of a turning point in the conventional wisdom about carcinogenesis. This can be seen in more recent research, for example:

Owens DM, Wei S, Smart RC. A multihit, multistage model of chemical carcinogenesis. Carcinogenesis, Vol. 20, No. 9, 1837-1844, September 1999

Abstract: Carcinogenesis involves the accumulation of genetic changes within a single cell. Tumor promotion functions in the initial clonal expansion of an initiated cell but is generally not considered to influence later stages. To investigate whether tumor promotion can influence later stages of carcinogenesis we developed a two-hit 7,12-dimethylbenz[a]anthracene (D) protocol designed to enrich for keratinocytes that contain at least two D-induced genetic alterations. FVB/N mice were initiated with D and promoted with 12-O-tetradecanoylphorbol-13-acetate (T) or treated with acetone (A) vehicle for 6 weeks. At 7 weeks after the start of promotion, but before visible papilloma development, groups of mice were treated with a second dose of D or A and 1 week later T promotion was resumed. D/T/A/T mice developed 2.8 papillomas/mouse and D/A/D/T mice demonstrated an additive tumor response and developed 5.8 papillomas/mouse. Importantly, D/T/D/T mice developed 12.4 papillomas/mouse, thereby demonstrating a synergistic tumor response compared with D/A/D/T and D/T/A/T mice. D/T/D/T papillomas exhibited increases in suprabasal S phase cells and keratin 13 expression when compared with D/T/A/T papillomas. D/T/D/T mice developed squamous cell carcinomas (SCCs) 10 weeks earlier than D/T/A/T mice and demonstrated a 96% malignancy incidence and 1.71 SCC/mouse compared with D/T/A/T mice, which demonstrated a 28% malignancy incidence and 0.32 SCC/mouse. Greater than 90% of D/T/A/T and D/T/D/T papillomas and SCCs contained mutant Ha-ras, while a normal Ha-ras allele persisted in all cases, indicating that a gene other than the remaining normal allele of Ha-ras was a target gene for the second D hit. These data demonstrate that: (i) promotion between the first and second hits has a profound outcome on carcinogenesis, presumably by increasing the probability that a second hit will occur in a previously initiated cell; (ii) continued promotion after the second hit is required for full expression of malignancy; (iii) the classic initiation–promotion protocol can be extended to a multihit, multistage model.

RFK Jr: The Real Story on Vaccines

AUGUST 15, 2019

Americans Can Handle an Open Discussion on Vaccines—RFK, Jr. Responds to Criticism from His Family

CHD NOTE: In early May 2019, Politico Magazine published an article written by three of Robert F. Kennedy, Jr.’s relatives, criticizing his advocacy for safe vaccines. After numerous requests, Politico magazine has refused to publish his response.

By Robert F. Kennedy, Jr.

Three of my Kennedy relatives recently published an article criticizing my advocacy for safe vaccines. Our contentious family dispute highlights the fierce national donnybrook over vaccinations that has divided communities and raised doubts about the Democratic Party’s commitment to some of its defining values: abhorrence of censorship, wariness toward excessive corporate power, support for free speech, religious freedom, and personal sovereignty over our bodies, and the rights of citizens (codified in the Nuremberg Code and other treaties to which we are signatories) to decline unwanted government-mandated medical interventions. The debate has also raised questions about the independence of our press and its role as a champion of free speech, and First Amendment rights as a bulwark against overreaching by government and corporations.

I love my family and sympathize with their anxieties when I call out government officials for corruption. The Kennedys have a long, close, and continuing relationship with public health agencies so it is understandably difficult for us to believe that powerful regulators would lie about vaccines. “All issues are simple,” the saw goes, “until you study them.” Read more …