The term “quackery” originated with the use of quicksilver (mercury) to treat patients. Along with blood letting, this was a common practice at one time. Both mercury and blood letting reduced the symptoms that the patient experienced by making the immune system so weak it could not respond. George Washington alleged died of this after being treated for catching a cold.

The dentists who continue to use mercury are the modern day “quacks” but what about physicians who use drugs that kill the patient rather than saving them.

Medical error is the third leading cause of death in the United States and the largest component of medical error is drugs administered to patients in hospitals. Most people are not aware of this or of the autopsy research that shows about 1/3 of patients that die in a hospital in the U.S. and the U.K. die of a cause that is not diagnosed and they are not being treated for.

Perhaps we could excuse the physicians who have a tough job if they were trying to do better. However, there has been no noticeable improvement in the medical error problem since I participated in the government investigation into this issue 13 years ago.

Jon Rappaport comments on his recent interview with Barbara Starfield who published epidemiological data on this problem in JAMA many years ago. Starfield’s work was reviewed over a decade ago on this blog.

THE STARFIELD REVELATION REVISITED

Fear of spiders, heights, flying, whatever? The first thing you should try is a simple process of tapping on acupuncture meridians. The reporter above had fear of spiders for 25 years and after a couple of minutes of tapping was able to hold a tarantulla and feel like it was cute and cuddly. See video …

For most emotional problems EFT is the first option of choice as it takes only a few minutes and has no negative side effects. It it works, great! If not, try something else. (Actually, if you persist with EFT and/or get some help it will always offer some benefit.)

Significant research material is available for EFT and you can download a manual for free.

During the 70s and 80s I spent 15 years as a leader and participant in various group therapy practices and found them useful. However, I was impressed at the amount of work and time it could take to eliminate small things and how many things after years of therapy were not possible to eliminate.

In frequency work I do not have the time or inclination to do therapeutic work. However, often people need some so I recommend a simple tapping technique which can sometimes do in a few minutes what cannot be done in years of therapy. Everyone should learn this as a child and use it regularly as needed. You can learn this on your own using information and videos available on the web, much of it for free.

Emotional Freedom Techniques (EFT) is a system of tapping on energy meridians in the upper part of the body while thinking about a negative event. Thinking about the event generates one set of signals in the brain and tapping generates another which disrupts the first.

Symptoms that people have from negative effects are caused by the signals in the brain that constantly regenerate and become more powerful by thinking about the event. The disruptive tapping signal disconnects the thinking signal from physical symptoms. Removing the reinforcing effect of the physical symptoms allows the repetitive signaling to fade away, along with the problem. This can often happen quite quickly in a few minutes.

Social Anxiety Solutions has multiple descriptions of how it works.

Mechanoreceptors are specialized receptors that respond to mechanical forces such as tapping, massaging, or holding. Among their types: Meissner corpuscles, Pacini corpuscles, Merkel discs, and Ruffini corpuscles.

They are sensitive to stimulation on the surface of the skin anywhere on the body.
The acupuncture points (the points tapped on with EFT), called “hsue” in traditional Chinese medicine, are loci that have a particularly high concentration of mechanoreceptors, free nerve endings, and neurovascular density.

The signals that are initiated when tapping hsue travel as afferent stimuli that are capable of reaching the cortex, the amygdala, and the hippocampus.

Mechanoreceptors are distributed all over the skin surface. The signal that is generated by tapping travels via large myelinated fibers, ascends ipsilaterally through the medial lemniscus, and triggers the somato-sensory cortex at the parietal lobes and the prefrontal cortex.

From there, the signal reaches the amygdala, hippocampus, and other structures where the emotional problem has neurological entity, and the signal apparently disrupts established patterns.

After EFT treatment there are reductions in cortisol, a primary stress hormone, and these reductions are accompanied by improvements in heart rate variability (HRV).

HRV and cortisol are primary stress markers for a wide variety of genetic, hormonal, and neurological effects of stress. They both correlate with significant changes in the conditions measured on the SA-45 questionnaire (Church, 2008c).

Salmonella paratyphi b

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Today, the Hawaii Department of Health announced a Salmonella paratyphi b outbreak linked to ahi tuna.  Interestingly, the ten illnesses in Hawaii appear to be linked to 13 other illnesses nationally, and possibly even linked to an outbreak of illnesses linked to the same product two years ago.  (We reported on this phase of the outbreak in February 2008).  Was it a frozen product that was held at the manufacturer or distributor’s facilities for two years, and finally released now?  Or is there a persistent source of the paratyphi b strain at the manufacturer’s facilities?

The paratyphi b strain of salmonella is different from those that we see most frequently in foodpoisoning outbreaks.  There are well over 2,000 individual strains of salmonella, but not all of them cause typhoid fever.  Paratyphi b, however, does cause typhoid fever. 

Typhoid fever is the most serious form of enteric fever, with humans being the sole reservoir of the bacteria. Based on a recent survey, the global number of typhoid cases in 2000 exceeded 21,000,000, with more than 200,000 deaths [1]. Enteric fever, that is typhoid and paratyphoid fevers, is the common name for infections caused by Salmonella enterica serotypes typhi and paratyphi. Of the three types of S. paratyphi (A, B, and C), B is the most common.

http://casesjournal.com/content/1/1/403.  Salmonella typhi and paratyphi are also more likely than other salmonella serotypes to cause enteric fever, as well as the chronic carrier state.

Typhoid fever, or enteric fever, differs from the typical salmonella gastrointestinal illness in that typhoid fever does not always cause severe gastrointestinal symptoms. 

The syndrome of enteric fever is characterized by prolonged sustained fever, relative bradycardia, hepatosplenomegaly, rose spots, and leucopenia and neutropenia [3]. After an incubation period of 5 to 21 days (generally 7 to 14 days), fever and malaise develop, often associated with cough. A small proportion of patients may have diarrhea during the incubation period. The fever tends to rise in stepwise fashion over the first few days to a week and then becomes sustained, usually at 39.4 to 40°C (103 to 104°F) or higher. After 2 weeks of illness, the severe complications of intestinal hemorrhage or perforation may be observed. The illness usually resolves by the end of the fourth week in an untreated patient. Relapse may occur in untreated as well as treated patients, but the illness is milder than the original episode. Rarely, some of the following complications may occur: pancreatitis, cholecystitis, infective endocarditis, pneumonia, hepatic or splenic abscess, orchitis, or focal infection at virtually any site [6].

The final step in migrating with Google to a new URL for Dr. Jeff Sutherland’s Electronic Medicine is to allow direct access at http://blog.frequencyfoundation.com.

Please update your URL for this blog to http://blog.frequencyfoundation.com

The (r)evolution of cancer genetics
Francesca D Ciccarelli
Department of Experimental Oncology, European Institute of Oncology, IFOM-IEO Campus, Via Adamello 16, 20139 Milan, Italy

BMC Biology 2010, 8:74doi:10.1186/1741-7007-8-74
The electronic version of this article is the complete one and can be found online at: http://www.biomedcentral.com/1741-7007/8/74

Recent advances in sequencing technologies and the launching of massive resequencing projects such as the Cancer Genome Project [1] have boosted the production of cancer genomics data. In the past few years, the entire repertoire of human exons has been sequenced in glioblastoma [2], pancreatic [3], breast and colorectal [4] cancers, and somatic mutations in selected genes have been mapped in multiple samples of renal [5] and lung [6] adenocarcinomas. In addition, the whole genomes of individuals affected by leukemia [7,8], melanoma [9], glioma [10], breast [11,12], and lung [13] cancers have been fully resequenced. All these studies have led to the identification of more than 1,000 potential cancer genes, and the list is likely to grow in the near future.

This massive amount of information will have a huge impact on our understanding of cancer genetics, even more so considering that the biological role of most mutations is still obscure. These first unbiased screenings have led to the identification of novel and unsuspected determinants of cancer, such as the isocitrate dehydrogenase enzyme genes IDH1 and IDH2, which have been found mutated in glioblastoma multiforme [2]. They have also started to question some cornerstones of cancer biology, such as the description of cancer as a unique disease driven by the somatic modification of a few key regulators. The progressive identification of novel mutated genes is expanding the ‘cast of actors’ [14] whose mutations might be causally involved in driving cancer. Moreover, given the high heterogeneity of genes mutated in different cancer types (Figure 1), the overall ‘plot’ is becoming more intricate. The emerging picture suggests that there may be distinct genetic routes to reach the common aftermath of all tumorigenic processes, which is uncontrolled cell proliferation. For example, as many as 12 core pathways are disrupted in the majority of pancreatic cancers through multiple somatic mutations [3]. This opens up an intriguing scenario where the deregulation of key pathways for tumorigenesis represents only the final step of a more general perturbation of cellular activity. The cell is seen as an integrated system in which all processes form a tightly interconnected network more than as an ensemble of independent pathways. In this context, the effect of somatic mutations occurring in the cancer genome should be interpreted in the light of their broader impact on the system’s equilibrium…

Mercury filings are toxic and outgassing whenever they are disturbed. The mercury goes to all organ systems in your body and reduces their performance and capability. Check out this video.